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Our study revealed pronounced oxidative stress in mice exposed to APAP, which contrasts with previous research showing that mimic peptides targeting necrotic cells in APAP-induced hepatotoxicity do not significantly alter liver oxidative status or GSH level (Maes et al

In conclusion, UPS has an important role in the development of AKI, mostly by NF-B, ferroptosis, oxidative stress, lipid homeostasis, and KLF4/ITCH/LATS1/YAP pathway

L., Witherden, D

As mentioned in the section above, IDH1 catalyzes typically the conversion of isocitrate to -KG, but the mutant isoform converts -KG into D-2HG instead, which has been shown to inhibit cellular differentiation through epigenetic alterations [19]