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Oral glutathione and growth in cystic fibrosis: a multicenter, randomized, placebo-controlled, double-blind trial

The mechanisms by which LA reduces liver fat content in HFD animals may be related to suppressing oxidative stress and lipid peroxidation, improving mitochondrial dysfunction, inhibiting insulin resistance, and regulating some related factors or genes (such as ROS-scavenging enzyme genes) to inhibit excessive fat synthesis in the liver

There might be some differences in oxidative stress status between bacterial and viral pneumonia