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In cultured human cells exposed to heavy metals (Zn, Co, Ni) known to induce DNA breaks and strand instability, Epitalon significantly reduced the extent of genotoxic damage, acting as a protective agent [36]

The mechanism likely involves the same angiogenic pathway: bone repair is acutely dependent on vascularization of the fracture gap, and BPC-157's VEGF-stimulating activity addresses this bottleneck directly

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