Considering the redox activity of potential anticancer Cu(II) chelates (Table 3 and references cited) [323,324,325,326,327] one possible consequence is that the high level of copper in proliferating tissue could also reduce oxidative redox potential which may in turn increase cancer cell proliferation [45, 50, 328, 329]
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No Human Pharmacokinetic Programme FDA found no pharmacokinetic data defining absorption, distribution, metabolism, half-life, clearance or tissue exposure for Epitalon free base or Epitalon acetate