Liu Y, Zhang Z, Li T, Xu H, Zhang H
34 Our data showed that BSO treatment indeed caused inhibition of the phosphorylation status of IkB in ESCC, suggesting that inhibition of the NF-kB signaling pathway in ESCC by BSO depends on IkB
Structure and Function of LGP2, a DEX(D/H) Helicase That Regulates the Innate Immunity Response
Many factors are presumably involved (Boogard et al., 1989), including relative distribution of N -acetyltransferase and aminoacylases, cellular and mitochondrial uptake mechanisms, susceptibility to inactivation of cysteine S -conjugate -lyases and nearby enzymes/macromolecules, relative K m values, competition from natural amino acid substrates, absolute level of the enzyme, the presence of the enzyme in mitochondria, and the presence of -keto substrate to keep the enzyme enzymatically active (if the lyase in question is an aminotransferase)