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In renal I/R injury, C3G increases GPX4 expression and GSH level, reverses excessive intracellular free iron accumulation, decreases lipid ROS, ACSL4, 4-HNE, and MDA levels, and significantly inhibits the ferroptosis of renal tubular cells

and the stem cell markers OCT4 (Forward: 5-GAGCCCTGCACCGTCACC-3 and Reverse: 5-TTGATGTCCTGGGACTCCTCC-3), SOX2 (Forward: 5-TACAGCATGTCCTACTCGCAGC-3 and Reverse: 5-GAGGAAGAGGTAACCACAGGGG-3), CXCR4 (Forward: 5-ACTACACCGAGGAAATGGGCT-3 and Reverse: 5-CCCACAATGCCAGTTAAGAAGA-3), and cMET (Forward: 5-AGCGTCAACAGAGGGACCT-3 and Reverse: 5-GCAGTGAACCTCCGACTGTATG-3) for 30 cycles using GoTaq qPCR Master Mix (Promega, Madison, WI, USA)

10.1021/jm00293a017 46 HeathH

SARAF degradation during pancreatitis leads to worsened disease severity, highlighting the delicate balance in calcium regulation mechanisms within acinar cells