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Additionally, MAPKs can activate nuclear factor kappa-B (NF-B), with the MAPK/NF-B pathway playing a crucial role in PF pathogenesis (Zhu et al., 2024)

In vivo studies have further demonstrated that PDA NPs can alleviate puncture-induced disc degeneration by suppressing ferroptosis and restoring antioxidant defenses, offering a novel therapeutic strategy for IDD that directly targets ferroptosis-related damage at the mitochondrial level (94,95)

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